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. 2021 Aug 10;38(1):47–68. doi: 10.1007/s12264-021-00759-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 9 — bMIPCs generate cortical OPCs and APCs. A-F EGFR, ASCL1 and OLIG2 triple immunostained GW18 and GW23 human cortical sections. tRGs that expressed EGFR and ASCL1 in the cortical VZ were shown (arrows in C, F). bMIPCs that expressed EGFR, ASCL1 and OLIG2 in the cortical IFL were shown (arrows in B, E). In the cortical IZ, OPCs (arrowheads) expressed ASCL1 and OLIG2, but downregulated EGFR expression, and APCs (arrows) expressed EGFR and OLIG2, but downregulated ASCL1 expression (A, D).