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. Author manuscript; available in PMC: 2022 Jan 22.
Published in final edited form as: Am J Physiol Cell Physiol. 2006 Feb 15;291(1):C83–C92. doi: 10.1152/ajpcell.00579.2005

Table 2.

Effect of [Mg2+]i on ICa in cardiac myocytes with PP2A and BAY K8644

Conditions ICa, pA/pF %Reduction of ICa
0.2 mM [Mg2+]p 1.8 mM [Mg2+]p
[Ca2+]o (2.0 mM) Control 17.0±2.0 (12) 5.6±1.0*(10) 64±2.8
PP2A 9.8±1.3 (12) 6.7±0.4*(16) 25±3.4
[Ca2+]o (0.5 mM) Control 8.0±0.7 (6) 1.9±0.3*(8) 75±2.4
BAY K8644 16.7±0.9 (4) 3.9±0.6*(4) 76±2.9
BAY K8644 + PP2A 11.6±1.4 (5) 2.5±0.3*(5) 75±2.9

Values are means ± SE. The numbers in parentheses indicate replicates for each experimental condition. ICa density in rat ventricular myocytes voltage-clamped with patch electrodes containing the indicated [Mg2+]p. [Ca2+]o (2.0 mM) values were from Ref. 37. Currents were recorded at 5 min after breakthrough into the whole cell patch-clamp configuration. PP2A was included in patch electrode solutions at a concentration of 5 U/ml. BAY K8644 (0.1 μM) was added to superfusion solutions with the indicated [Ca2+]o.

*

P < 0.05, statistically significant changes of ICa between 0.2 and 1.8 mM [Mg2+]p.

P < 0.05, a reduction of ICa by [Mg2+]i that is significantly different than that produced under control conditions.

P < 0.05, statistically significant difference between control and experimental condition at the same [Mg2+]p.