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. 2022 Jan 10;8:814951. doi: 10.3389/fmed.2021.814951

Figure 2.

Figure 2

Associations between liver fatty acids and significant liver fibrosis. Data are presented as adjusted odds ratios (OR) with 95% confidence intervals (CI) and P-values for each standard deviation change in liver fatty acid proportions. OR are adjusted for BMI, age and PNPLA3 (I148M) genotype. 14:0, myristic acid; 15:0, pentadecaenoic acid; 16:0, palmitic acid; 16:1n-7, palmitoleic acid; 17:0, heptadecaenoic acid; 18:0, stearic acid; 18:1n-9, oleic acid; 18:1n-7, vaccenic acid; 18:1, oleic acid combined with vaccenic acid; 18:2n-6, linoleic acid; 18:3n-6, γ-linolenic acid; 18:3n-3, α-linolenic acid; 20:0, arachidic acid; 20:3n-6, dihomo-γ-linolenic acid; 20:4n-6, arachidonic acid; 20:5n-3, eicosapentaenoic acid; 22:0, behenic acid; 22:5n-3, docosapentaenoic acid; 22:6n-3, docosahexaenoic acid; 24:0, lignoceric acid; SCD-1, stearoyl-coA desaturase; D5D, delta 5 desaturase; D6D, delta 6 desaturase; AA/LA, arachidonic acid/linoleic acid. SCD-1, D5D and D6D are estimated using fatty acid product-to-precursor ratios: 16:1n-7/16:0 (SCD-1), 20:4n-6/20:3n-6 (D5D), 18:3n-6/18:2n-6 (D6D). D5D in cholesteryl esters: n(F0–1) = 19, n(F2–4) = 16 due to 25 zero-values of 20:3n-6.