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. 2022 Jan 25;185(6):1025–1040.e14. doi: 10.1016/j.cell.2022.01.018

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© 2022 The Authors

Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active.

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Figure S5 — Anti-SARS-CoV-2 RBD IgG signatures following BNT162b2 vaccination and SARS-CoV-2 infection, related to Figure 5

(A) Ratios of anti-Wuhan-Hu-1 to variant RBD IgG concentration are shown for Stanford individuals who received BNT162b2 vaccination at different time points after the second dose (D21, n = 58 individuals) and third dose (around month 9, n = 36 individuals) vaccination. Box-whisker plots show the interquartile range as the box and the whisker ends as the most extreme values within 1.5 times the interquartile range below the 25% quantile and above the 75% quantile.

(B) Principal component analysis (PCA) of anti-SARS-CoV-2 Wuhan-Hu-1 and viral variant RBD IgG concentrations across Stanford BNT162b2 vaccinees, Stanford COVID-19 patient cohort 2, and SARS-CoV-2-variant-infected patients.