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. 2021 Oct 20;59(1):276–293. doi: 10.1007/s12035-021-02591-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Enriched disease pathway networks in female and male neurons and microglia. AD compared to non-AD functionally enriched pathways with a BH-adjusted p value < 0.05 clustered into biological themes for a. excitatory and b. inhibitory neurons from the prefrontal cortex; c. neurons from the entorhinal cortex; and microglia from the d. prefrontal and e. entorhinal cortices. Lines represent gene set overlaps with magnitude showed by thickness