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. 2022 Jan 19;2022:1182384. doi: 10.1155/2022/1182384

Table 1.

Patient characteristics.

Clinical characteristics Patient 1 Patient 2 Patient 3 Patient 4
Age/sex 70s/M 60s/F 90s/F 60s/M
Initiation of antilymphoma treatment First line: rituximab and acalabrutinib First line: ibrutinib and rituximab Acalabrutinib 1st line: VDR
2nd line: ibrutinib + rituximab
Clinical presentation leading to the diagnosis Symptomatic hepatomegaly, diastolic CHF Stage IV CKD, anemia, no significant proteinuria Transfusion dependent anemia, CHF, stage IV CKD Dyspnea on exertion, lymphadenopathy
Circulating monoclonal protein (g/dL) IgM kappa, M spike 2.6 IgM lambda, M spike 0.4 IgM kappa, M spike 2.2 IgM lambda, M spike 4
IgM (40–230 mg/dL) 2666 452 2949 8020
Serum-free kappa (3.30–19.40 mg/L) 205 30 705 7
Serum-free lambda (5.7–26.3 mg/L) 3 39 101 170
Kappa to lambda ratio 68 0.81 7 0.04
dFLC at presentation 202 9 604 163
Troponin T (0.000–0.029 ng/mL) 0.042 Not tested 0.1 0.047
NT-proBNP (0–450 pg/mL) 4928 650 10500 2300
Cardiac modified Mayo stage (2015) IIIa N/A IIIb IIIa
Serum albumin (3.6–5.1 g/dL) 4.2 3.9 3.1 3.9
Creatinine (0.58–0.96 mg/dL) 1 2.41 2.5
Cholesterol (normal is <200 mg/dL) 182 198 185 144
Alkaline phosphatase (33–130 U/L) 407 99 75
24-hour urinary protein (normal is <200 mg) 150 191 99 130
ECHO findings Grade III left ventricular diastolic dysfunction, LVEF 48%, mild upper septal left ventricular hypertrophy Normal LV and RV, LVEF 58%, grade I left ventricular diastolic dysfunction Grade II left ventricular diastolic dysfunction, LVEF 28%, there is mild upper septal left ventricular hypertrophy Grade III left ventricular diastolic dysfunction, LVEF 57%, RVH and LVH, advanced cardiac amyloidosis with restrictive physiology, sparkling granular appearance, and apical sparing
Tissue biopsy confirming the diagnosis of AL amyloidosis Liver, bone marrow, and cardiac biopsy Renal biopsy Bone marrow biopsy Lymph node biopsy showing both LPL and amyloid
LC-MS/MS analysis The main amyloidogenic component is kappa immunoglobulin light chains The main amyloidogenic component is lambda immunoglobulin light chains Amyloid type confirmed with immunohistochemistry Amyloid type confirmed with immunohistochemistry
Bone marrow biopsy findings 40–50% involvement by LPL, amyloid + 50% involvement by LPL, amyloid − 90% involvement by LPL, gain of chromosomes 4 and 18 Not done
MYD88 status Mutated Mutated Mutated Mutated
Complications during treatment with BTK-I and rituximab Rituximab flare, thumb hematoma (Figure 4) leading to 50% dose reduction of acalabrutinib None None Atrial fibrillation leading to discontinuation of ibrutinib
Antilymphoma therapy prior to initiating BTK inhibitor-based regimen None None Intolerance to rituximab and bortezomib First line (11/2016–3/2017): VDR with PR
2nd line (4/2017–12/2017): ibrutinib + rituximab with VGPR
3rd line (1/2018–6/2018): BR with stable disease
4th line: antiamyloid fibril MAB + CyBorD in a clinical trial with VGPR
Best hematologic response/outcome with BTK inhibitor therapy/time to response VGPR with hepatic and cardiac response/8 months CR/12 months VGPR/10 months CR/9 months
Organ response/time to response Cardiac/hepatic response/6 months Stable disease without renal progression to date Cardiac response/6 months
Stable disease without renal progression
Cardiac response/6 months

BR: bendamustine and rituximab, BTK-I=Bruton's tyrosine kinase inhibitor, CKD: chronic kidney disease, CHF: congestive heart failure, dFLC = difference in free light chain levels, CR: complete response, LC-MS/MS: liquid chromatography with tandem mass spectrometry, CyborD: cyclophosphamide, bortezomib, and dexamethasone, LPL: lymphoplasmacytic lymphoma, VGPR: very good partial response, VDR: bortezomib, lenalidomide, and dexamethasone, LV = left ventricle, RV = right ventricle, LVEF = left ventricular ejection fraction, LVH = left ventricular hypertrophy, RVH = right ventricular hypertrophy, MAB = monoclonal antibody, and ECHO = echocardiogram.