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. 2022 Jan 26;22:40. doi: 10.1186/s12935-022-02451-y

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Specific sequencing data of 20 patients with MDS. a Summary of mutation data in the MDS cohort. b The bar plot shows the number of specific mutations in our MDS cohort. c Functional structures and mutations of ASXL1 and U2AF1 proteins. ASX: Asx homology, ASXN: Asx N-terminal, NR box: nuclear receptor co-regulator binding motif, PHD: plant homeodomain, ZNF: zinc finger, UHM: U2AF homology motif.