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. 2021 Oct 29;46(2):333–341. doi: 10.1038/s41366-021-01005-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — AGO2 communicates with DELTA133P53, CCNE1 and STAT3 in obese patients to potentially mediate increased CRC risk via increased inflammation. AGO2 communicates with DELTA133P53 and CCNE1 in non-obese patients following RYGB to potentially mediate reduced CRC risk via reduced inflammation.