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The pathway of diabetes pathogenesis. IAP, intestinal alkaline phosphatase; IL-1β, interleukin-1β; IRS, insulin receptor substrate; JNK, c-Jun N-terminal kinases; LPS, lipopolysaccharides; LTA, lipoteichoic acids; NF-kB, nuclear factor-kB; NSAID, non-steroidal anti-inflammatory drugs; TNF-α, tumor necrosis factor-α.
