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. 2022 Jan 28;17:48. doi: 10.1186/s13018-021-02886-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — miR-155 was highly expressed in LPS-induced mouse KOA chondrocytes. The mouse knee chondrocyte inflammatory model was established by LPS induction. A The cell morphology was observed using an inverted microscope; B The cell morphology was observed using toluidine blue staining; C The expression of the knee chondrocyte marker Collagen II was detected by immunocytochemistry; D The expression of miR-155 was detected using RT-qPCR. The cell experiment was repeated 3 times. The data were expressed as mean ± standard deviation. Independent t test was used for data comparisons between 2 groups. *** P < 0.001