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. 2022 Jan 7;23(3):205. doi: 10.3892/etm.2022.11128

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Copyright: © Wei et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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WG ameliorates DOX-induced myocardial injury in rats. WG improved the damage induced by DOX in rats as shown by of (A) echocardiography and (B) ECG. Specifically, WG reversed the decrease of (C) EF and (D) FS, the increase of (E) R wave and (F) T wave and the prolongation of (G) QT interval in DOX rats. In addition, WG also improved the contents of serum (H) cTnT, (I) CK-MB and (J) BNP in DOX rats. All values are the mean ± SD. ^*P<0.05, ^**P<0.01 vs. CON; ^#P>0.05, ^##P>0.01 vs. DOX. WG, wogonin; DOX, doxorubicin; ECG, electrocardiograms; EF, ejection fraction; FS, fractional shortening; CON, control; cTnT, cardiac troponin T; CK-MB, creatine kinase MB; BNP, brain natriuretic peptide.