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. 2022 Jan 7;23(3):205. doi: 10.3892/etm.2022.11128

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Copyright: © Wei et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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WG improves the DOX-induced oxidative stress and apoptosis in rat hearts. WG reversed the decrease of (A) SOD, (C) GSH and (D) CAT and the increase of (B) MDA in myocardial tissue of DOX rats. (E) WG also improved cardiomyocyte apoptosis in DOX rats (magnification, x200). (F) Hematoxylin and eosin staining (magnification, x200). (G) Quantitative analysis. All values are the mean ± SD. ^**P<0.01 vs. CON; ^#P>0.05, ^##P>0.01 vs. DOX. WG, wogonin; DOX, doxorubicin; SOD, superoxide dismutase; GSH, glutathione; CAT, catalase; MDA, malondialdehyde.