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. 2019 Oct;8(6):2357–2370. doi: 10.21037/tcr.2019.09.59

Table 2. Comparison of ORRs and DCRs between 48 mCRC patients with wild-type RAS and 25 mCRC patients with mutant-type RAS in the two groups of second-line treatment.

Variable Wild type of RAS gene Mutant type of RAS gene
mFOLFOX6, (N=32) (%) mFOLFOX6 + VEGFi, (N=16) (%) P value mFOLFOX6, (N=16) (%) mFOLFOX6 + VEGFi, (N=9) (%) P value
Response 0.003 0.030
   PR 0 (0) 3 (18.8) 0 (0) 2 (22.3)
   SD 10 (31.3) 9 (56.2) 3 (18.8) 4 (44.4)
   PD 22 (68.7) 4 (25.0) 13 (81.2) 3 (33.3)
Responder 0.011 0.049
   Yes 0 (0) 3 (18.8) 0 (0) 2 (22.3)
   No 32 (100.0) 13 (81.2) 16 (100.0) 7 (77.7)
DCR 0.004 0.017
   Yes 10 (31.3) 12 (75.0) 3 (18.8) 6 (66.7)
   No 22 (68.7) 4 (25.0) 13 (81.2) 3 (33.3)

ORR, objective response rate; DCR, disease control rate; mCRC, metastatic colorectal cancer; VEGFi, vascular endothelial growth factor inhibitors (aflibercept and bevacizumab); PR, partial response; SD, stable disease; PD, progressive disease. Responder: yes, partial response; no, stable disease + progressive disease.