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. 2022 Jan 31;22:124. doi: 10.1186/s12885-021-09005-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Overexpressed miR-503-5p inhibits the viability, migration and invasion, and induces apoptosis of SK-OV-3 cells. A The expression of miR-503-5p after transfection of miR-NC/ miR-503-5p mimics or inhibitor NC/miR-503-5p inhibitor into SK-OV-3 cells was detected by qRT-PCR. ***P < 0.001 vs. the miR-NC group, ###P < 0.001 vs. the inhibitor NC group. B-E The viability, migration, invasion and apoptosis of SK-OV-3 cells transfected with miR-503-5p mimics/miR-NC was measured by MTT assay, wound healing assay, invasion assay and flow cytometry, respectively. **P < 0.01 vs. the miR-NC group