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. 2022 Jan 5;13(1):146–150. doi: 10.1055/s-0041-1740611

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Association for Helping Neurosurgical Sick People. This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. ( https://creativecommons.org/licenses/by-nc-nd/4.0/ )

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 1 — ( A ) MRI brain T2 weighted image (WI) showing symmetric hyperintensities in bilateral dentate nuclei in isoniazid toxicity. ( B ) MRI brain T2WI showing symmetric hyperintensities in bilateral dentate nuclei in ornidazole toxicity. ( C ) MRI brain FLAIR image showing hyperintensities in bilateral dentate nuclei, middle cerebellar peduncle, and pons in acute disseminated encephalomyelitis. ( D ) MRI brain T2WI showing symmetric hyperintensities in bilateral dentate nuclei and cerebellar hemispheres in cerebrotendinous xanthomatosis. Bilateral cerebellar hemispheres have prominent folia suggestive of cerebellar atrophy.