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. 2022 Jan 5;13(1):146–150. doi: 10.1055/s-0041-1740611

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Association for Helping Neurosurgical Sick People. This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. ( https://creativecommons.org/licenses/by-nc-nd/4.0/ )

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 2 — ( A ) MRI brain T2 weighted image (WI) showing symmetric hyperintensities in bilateral dentate nuclei and left middle cerebellar peduncle in Neurofibromatosis type 1. ( B ) CT brain showing hyperdensities in bilateral dentate nuclei and cerebellar hemispheres in primary hypoparathyroidism. ( C ) CT brain showing hyperdensities in bilateral basal ganglia and supratentorial white matter in primary hypoparathyroidism. ( D ) MRI brain T2WI showing symmetric hyperintensities in bilateral dentate nuclei, middle cerebellar peduncle, and pons in chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids.