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. 2022 Jan 31;12:1593. doi: 10.1038/s41598-022-05653-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of licochalcone A on LPS-induced protein levels of peroxisome proliferator-activated receptor γ (PPARγ), p-κB inhibitor α (IκBα) and p-p65 in claw dermal cells after treatment for 12 h (a,b), 24 h (c,d) and 48 h (e,f). Cells were exposed to gradient concentrations of licochalcone A (1, 5, 10 μg/mL) with or without the presence of 10 μg/mL LPS for 12, 24, and 48 h, respectively. The relative protein expressions of PPARγ/β-actin, p-IκBα/β-actin and p-p65/p65 were measured. The data were presented as mean ± SEM (n = 6). ^##P < 0.01 vs. control group; ^#P < 0.05 vs. control group; *P < 0.05 vs. LPS model group; **P < 0.01 vs. LPS model group (images are cropped. The full-length blots are presented in Supplementary file).