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. 2020 Dec 31;12(1):196–207. doi: 10.1080/21655979.2020.1860492

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© 2020 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 4. — PGM5P4-AS1 inhibited the migration and invasion activities of lung cancer cells. (a), The migration ability of A549 cells was assessed by wound healing assay, and the results showed that interference in the expression of PGM5P4-AS1 could promote cell migration (×100, *p < 0.05 vs. NC siRNA). The scale bar represented 200 μm. (b), The results of the migration ability of SK-MES-1 cells showed that the overexpression of PGM5P4-AS1 could inhibit cell migration (×100, #p < 0.05 vs. vector). The scale bar represented 200 μm. (c), Invasion abilities were measured by transwell assay, the results showed that interference in the expression of PGM5P4-AS1 could promote cell invasion (×200, *p < 0.05 vs. NC siRNA). The scale bar represented 100 μm. (d), The overexpression of PGM5P4-AS1 could inhibit the cell invasion (×200, #p < 0.05 vs. vector). The scale bar represented 100 μm