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. 2021 Sep 13;12(1):6403–6417. doi: 10.1080/21655979.2021.1971508

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© 2021 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 6. — SATB1-AS1 knockdown stimulates the repressive role of GSK3β on β-catenin expression. (a) The expression of GSK3β and β-catenin in parental and drug-resistance cells detected by immunoblotting. (b) The expression of GSK3β and β-catenin in parental cells overexpressing SATB1-AS1 and drug-resistance cells with SATB1-AS1 knockdown detected by immunoblotting. (c) The expression of GSK3β and β-catenin in HL60/Adr cells and OCI-AML5/Cty cells transfected with miR-580 inhibitor or oe-OAS2 in the presence of SATB1-AS1 knockdown detected by immunoblotting. Each assessment was done in triplicate with 3-time repetition to ensure minimum deviation; statistical data were measurement data, and described as mean ± standard deviation; Two-way ANOVA was applied for multiple-group comparisons, followed by Tukey’s multiple comparisons test. * p < 0.05