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. 2022 Jan 20;25(3):96. doi: 10.3892/mmr.2022.12612

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Hydrogen sulfide reduces the release of inflammatory factors and apoptosis in mice with LPS-induced acute kidney injury. (A) IL-6, (B) TNF-α, (C) IL-1β and (D) IL-18 levels in serum. (E) IL-1β and (F) IL-18 levels in kidney tissues. (G) TUNEL staining shows the level of apoptosis in kidney tissue. Scale bar, 20 µm. (H) Statistics of apoptosis index in kidney tissue of each group. Data are presented as the mean ± SD (n=10). LPS, lipopolysaccharide; IL, interleukin; TNF-α, tumor necrosis factor-α; NaHS, sodium hydrosulfide hydrate.