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. 2022 Feb 2;13(2):99. doi: 10.1038/s41419-022-04575-5

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — (A) Detection of TKT expression in 10 pairs of colorectal cancer CRC and adjacent tissues. (B) In the Gene Expression Profiling Interactive Analysis (GEPIA), TKT expression was high in CRC. (C) TKT immunostaining in TMAs. Note: magnification ×100. (D) Distribution of TKT staining intensity difference in tumor and tumor-adjacent tissues. Note: N, tumor-adjacent tissues; C, tumor tissues, P < 0.001. (E) High TKT expression is regulated to poorer overall survival of patients with CRC (P < 0.001). (F) High TKT expression is regulated to poorer disease-specific survival of colorectal carcinoma patients (P < 0.001, log-rank test).