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. 2022 Feb 3;13:8. doi: 10.1007/s12672-022-00469-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Knockdown of CENPO inhibited tumor growth in mice xenograft models. The mice xenograft model was established to observe the effects of CENPO knockdown on fluorescence expression intensity (A), tumor volume (B) and weight (C, D). E The expression of CENPO, Ki67, AKT and p-AKT was detected by IHC staining in mouse tumor tissues of shCENPO group and shCtrl group. The presented results were representative of experiments repeated at least three times. Data was represented as mean ± SD. *P < 0.05, **P < 0.01, ***P < 0.001