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. 2022 Feb 3;13(2):109. doi: 10.1038/s41419-022-04542-0

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — HNSCC cells (CAL-27) were treated with sh-LINC01123, OE-LINC01123, miR-214-3p mimic, miR-214-3p inhibitor, or sh-B7–H3. A Cell Counting Kit 8 (CCK8) assay of the proliferation of HNSCC cells. B Motility of HNSCC cells was determined by scratch wound healing assay. C Statistical results of scratch wound healing assay. D Migration and invasion of HNSCC cells were determined by transwell migration and invasion assay. E Statistical analysis of transwell results. F Apoptosis rate of HNSCC cells, as detected by flow cytometry. G Statistical results of the apoptosis rate. H Cell cycle stages of HNSCC cells, as detected by flow cytometry. I Statistical analysis of the cell-cycle results. *P < 0.05 versus the blank group. The experiment was repeated three times.