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. 2022 Jan 21;13:809433. doi: 10.3389/fnagi.2021.809433

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Copyright © 2022 Cai, Chai, Fu, Wang, Zhang, Zhang, Zhu, Miao and Yan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Sal mitigated pyroptosis in Aβ1-42 and D-gal/AlCl₃-induced AD mice. Aβ1-42 and D-gal/AlCl₃-induced AD mice brain expression of IL-1β (A) and IL-18 (B) analyzed by enzyme-linked immunosorbent assay (ELISA) kits (n = 4). (C) The Western blot analysis of IL-1β, IL-18, and cleaved GSDMD in the hippocampus of the AD mice (n = 3). Image J was applied to quantify the bands, and a histogram represents the differences after normalization to β-actin. All data are represented as mean ± SEM. ^##P < 0.01, ^###P < 0.001 vs. control group, ^*P < 0.05, ^**P < 0.01, vs. Aβ1-42 and D-gal/AlCl₃ group.