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. 2022 Jan 8;298(2):101572. doi: 10.1016/j.jbc.2022.101572

Figure 1.

Figure 1

Peroxisomal β-oxidation was enhanced in livers of the STZ-induced diabetic mice.A, gene expressions of enzymes in peroxisomal β-oxidation were upregulated in livers of the STZ-induced diabetic mice. B, peroxisomal β-oxidation was enhanced in liver of the STZ-induced diabetic mice. C, plasma glucose was elevated remarkably in the STZ-induced diabetic mice. D, liver-free fatty acids increased significantly in the STZ-induced diabetic mice. E, liver LC-acyl-CoA increased significantly in the STZ-induced diabetic mice. ABCD1, peroxisomal ATP-binding cassette transporter D; ACOX1, acyl-CoA oxidase-1; ACS, acyl-CoA synthetase; ACSL1, long-chain acyl-CoA synthetase-1; L-BP, L-bifunctional protein; STZ, streptozotocin. Mean ± SEM, n = 6, ∗p < 0.05 by t test between paired conditions.