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. 2022 Feb 7;21:37. doi: 10.1186/s12943-022-01517-9

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — TCGA bladder cancer cohort analysis on HNRNPU expression and its impact on survival. (A) Expression profile of HNRNPU in bladder tumors versus control tissues in TCGA dataset. (B) Pan-cancer analysis of HNRNPU expression in multiple cancer types. (C) Promoter methylation level of HNRNPU in TCGA dataset (D). Disease free survival analysis of high HNRNPU patients versus low HNRNPU patients. (E) Overall survival analysis of high HNRNPU patients versus low HNRNPU patients (high = top 20%). (F) Co-expression correlation between HNRNPU and HNRNPK. (G) Co-expression correlation between HNRNPU and AHCTF1; R = Pearson’s r (H) Overall survival analysis between AHCTF1 high patients versus AHCTF1 low patients (high = top 50%). (I) Expression profile of HNRNPU in TCGA bladder cancer immune cell subsets. GEPIA2021 was used to generate the plot