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. 2022 Jan 27;414(6):2235–2250. doi: 10.1007/s00216-021-03860-0

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Scatterplots of enriched KEGG pathways for combined lipidomic/metabolomic experiments when comparing Pex11α knockout (KO; Pex11α^−/−) with wild type (WT; Pex11α^+/+) control mice. Pathway topology analysis depicting the dysregulated metabolic pathways in a serum, b liver, and c heart tissue homogenates, respectively. Here, the x-axis represents the pathway impact, and the y-axis represents the pathway enrichment. Each node marks a pathway. The size (pathway impact value) and color (-log10 p-value) of the nodes represent the number of lipid/polar metabolite species and level of significance, respectively