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. 2022 Feb 8;28:19. doi: 10.1186/s10020-022-00444-1

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — UA inhibited proliferation, migration and invasion of TGF-β1-stimulated CFs through activating Nrf2. A and C The mRNA level of Nrf2 in CFs after indicated treatments. B and D The protein level of Nrf2 in CFs after indicated treatments. β-actin was used as normalized control. E Cell viability was measured by MTT assay of CFs after indicated treatments. F Cell migration was measured by wound healing assay of CFs after indicated treatments. G Cell invasion ability was measured by transwell assay of CFs after indicated treatments. N = 3, *P < 0.05, **P < 0.01, ***P < 0.001