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. 2022 Feb 8;28:19. doi: 10.1186/s10020-022-00444-1

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Nrf2 pathway was responsible for UA-induced repression of CMT process in CFs with TGF-β1 treatment. A The mRNA level and B the protein level of fibrosis markers including vimentin, DDR2, tensin and α-SMA in CFs after indicated treatments. C The protein levels of Nrf2, SOD1, HO-1 and NQO1 in CFs after indicated treatments. β-actin was used as normalized control. NC: negative control. N = 3, *P < 0.05, **P < 0.01, ***P < 0.001