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. 2022 Feb 10;21:43. doi: 10.1186/s12943-022-01519-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — METTL7B promoted glutathione metabolism in LUAD cells. Untargeted metabolomic analysis by UPLC-MS/MS was detected in PC9 cells stably transfected with FLAG-NC or FLAG-METTL7B. a Heat map of differential metabolites. b KEGG pathway of differential metabolites. c and d ROC analysis for reduced glutathione and oxidized glutathione. e-i ROS level was detected in METTL7B-overexpressed PC9, HCC827 and METTL7B-suppressed PC9-GR, H1975 and PC9-OR cells. ***P < 0.001 and ****P < 0.0001