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. 2022 Feb 12;22:165. doi: 10.1186/s12885-022-09265-1

Table 2.

Association of ESR1 fusions with clinical parameters in the predictive cohort

Predictive Endocrine Therapy Cohorts
Parameters n at least one ESR1-CCDC170
(exon 2 to 8) fusion
P-Value ESR1-CCDC170 (exon 2) fusion PValue ESR1-CCDC170 (exon 8) fusion P-Value ESR1-AKAP12 P-Value
n % n % n % n %
All patients 322 89 27.6% 50 15.5% 51 15.8% 13 4.0%
Age at start 1st line treatment (years)
  ≤ 50 63 19 30.2% 0.63 12 19.0% 0.62 8 12.7% 0.029 1 1.6% 0.36
   > 50- ≤ 70 161 37 23.0% 23 14.3% 24 14.9% 7 4.3%
   > 70 98 33 33.7% 15 15.3% 19 19.4% 5 5.1%
Menopausal status at start of 1st line treatment
  Premenopausal 64 17 26.6% 0.82 10 15.6% 0.99 8 12.5% 0.41 1 1.6% 0.26
  Postmenopausal 257 72 28.0% 40 15.6% 43 16.7% 12 4.7%
Surgery type
  Lumpectomy 95 25 26.3% 0.79 14 14.7% 0.90 15 15.8% 0.83 2 2.1% 0.89
  Ablation 169 42 24.9% 24 14.2% 25 14.8% 4 2.4%
Radiotherapy
  No 105 30 28.6% 0.33 20 19.0% 0.08 16 15.2% 0.98 2 1.9% 0.74
  Yes 159 37 23.3% 18 11.3% 24 15.1% 4 2.5%
Nodal status
  No lymph nodes 122 33 27.0% 0.88 19 15.6% 0.99 20 16.4% 0.95 4 3.3% 0.2
  Positive lymph nodes 130 38 29.2% 21 16.2% 22 16.9% 9 6.9%
  Tumor outside lymph nodes 53 15 28.3% 8 15.1% 7 13.2% 0 0.0%
  Not applicable (M1) 16 3 18.8% 2 12.5% 2 12.5% 0 0.0%
Pathological Tumor classification
  pT1 85 22 25.9% 0.60 13 15.3% 0.21 14 16.5% 0.90 2 2.4% 0.36
  pT2 + unknown 186 50 26.9% 25 13.4% 30 16.1% 10 5.4%
  pT3 + pT4 51 17 33.3% 12 23.5% 7 13.7% 1 2.0%
Tumor grade
  Poor 160 45 28.1% 0.36 27 16.9% 0.60 27 16.9% 0.60 7 4.4% 0.078
  Unknown 81 18 22.2% 10 12.3% 10 12.3% 0 0.0%
  Moderate/Good 74 24 32.4% 13 17.6% 13 17.6% 5 6.8%
Tumor cell content
  30–49% 27 7 25.9% 0.96 4 14.8% 0.99 2 7.4% 0.25 2 7.4% 0.63
  50–70% 98 28 28.6% 15 15.3% 13 13.3% 4 4.1%
   > 70% 197 54 27.4% 31 15.7% 36 18.3% 7 3.6%
Hormone/ growth factor status (RT-qPCR)
  ESR1-negative 0 0 0 0 0
  ESR1-positive 322 89 27.6% 50 15.5% 51 15.8% 13 4.0%
  PR-negative 63 18 28.6% 0.87 11 17.5% 0.65 11 17.5% 0.70 6 9.5% 0.014
  PR-positive 258 71 27.5% 39 15.1% 40 15.5% 7 2.7%
  HER2 non-amplified 279 77 27.6% 0.63 44 15.8% 0.85 45 16.1% 0.81 13 4.7% 0.16
  HER2 amplified 41 12 29.3% 6 14.6% 6 14.6% 0 0.0%
  CCDC170 negative 31 5 16.1% 0.13 2 6.5% 0.15 4 12.9% 0.62 0 0.0% 0.23
  CCDC170 positive 287 83 28.9% 47 16.4% 47 16.4% 13 4.5%
Adjuvant endocrine therapy
  No 252 66 26.2% 0.24 38 15.1% 0.64 36 14.3% 0.13 7 2.8% 0.030
  Yes (AI cohort only) 69 23 33.3% 12 17.4% 15 21.7% 6 8.7%
Adjuvant chemotherapy
  No 267 76 28.5% 0.47 40 15.0% 0.55 45 16.9% 0.27 12 4.5% 0.36
  Yes 55 13 23.6% 10 18.2% 6 10.9% 1 1.8%
Disease-free interval
   ≤ 1 year disease-free 72 23 31.9% 0.47 14 19.4% 0.62 12 16.7% 0.99 2 2.8% 0.45
  1–3 years disease-free 137 37 27.0% 20 14.6% 20 14.6% 8 5.8%
   > 3 years disease-free 113 29 25.7% 16 14.2% 19 16.8% 3 2.7%
Dominant site of metastasis
  Local regional 29 10 34.5% 0.51 7 24.1% 0.32 4 13.8% 0.36 0 0.0% 0.40
  Bone 159 40 25.2% 25 15.7% 21 13.2% 6 3.8%
  Other distant metastasis 130 38 29.2% 17 13.1% 25 19.2% 7 5.4%
Response type
  Complete response 11 3 27.3% 0.87 2 18.2% 0.73 1 9.1% 0.29 0 0.0% 0.46
  Partial response 39 9 23.1% 3 7.7% 6 15.4% 2 5.1%
  Stable disease over 6 months (SD > 6 m) 115 32 27.8% 16 13.9% 23 20.0% 1 0.9%
  Stable disease for 6 months or less (SD ≤ 6 m) 13 2 15.4% 2 15.4% 1 7.7% 0 0.0%
  Progressive disease (PD) 83 20 24.1% 14 16.9% 8 9.6% 3 3.6%
Response type
  No response 96 22 22.9% 0.50 16 16.7% 0.38 9 9.4% 0.05 3 3.1% 0.50
  Response 165 44 26.7% 21 12.7% 30 18.2% 3 1.8%

ESR1 estrogen receptor alpha, CCDC170 coiled-coil domain containing 170, AKAP12 A-Kinase Anchoring Protein 12 gene, ESR1-CCDC170 ESR1-CCDC170 gene fusion, ESR1-AKAP12 ESR1-AKAP12 gene fusion, M1 methastatic stage 1, pT primary tumor, pT1 small primary tumor (tumour is 2 cm across or less), pT2 tumour more than 2 cm but no more than 5 cm across, pT3 T3 tumour bigger than 5 cm across, pT4 tumor with phatological stage, RT-qPCR Quantitative Real-Time Polymerase Chain Reaction, PR progesterone receptor, HER2 human epidermal growth factor receptor, AI aromatase inhibitors, SD standard deviation, PD progressive disease

Statistically significant differences are indicated in bold