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. 2022 Jan 19;14(3):431. doi: 10.3390/nu14030431

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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(A) Catalase (C = 8; CCr = 8; D = 7; DCr = 6), (B) superoxide dismutase (C = 5; CCr = 6; D = 6; DCr = 5) and (C) glutathione peroxidase renal activity (C = 6; CCr = 6; D = 7; DCr = 4). (D) Hydrogen peroxide content (C = 4; CCr = 4; D = 5; DCr = 5), (E) thiobarbituric acid reactive substances (C = 6; CCr = 6; D = 5; DCr = 6), (F) carbonylated protein content (C = 5; CCr = 4; D = 4; DCr = 4) renal of the experimental groups at the end of the experimental protocol. The graph represents mean ± SEM. The divergent letters represent mean significant differences compared to the control group. (A) b p < 0.01; (B) b p < 0.01; (C) b p < 0.001; (D) b p < 0.01. (E) b p < 0.001. (F) b p < 0.05. C—normoglycemic non-supplemented animals; CCr—normoglycemic creatine supplemented animals; D—non-supplemented STZ-induced diabetics animals; DCr—creatine-supplemented STZ-induced diabetics animals.