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. 2022 Feb 5;8(2):e08904. doi: 10.1016/j.heliyon.2022.e08904

Figure 4.

Figure 4

Inflow of B cells in pulp during chronic systemic inflammation. (A) Percentages of common myeloid progenitor cells (CMPs), granulocyte-monocyte progenitor cells (GMPs), and megakaryocyte-erythroid progenitor cells (MEPs) in BM, DP, and PB of poly(I:C)-treated mice and non-treated mice 24 h after chronic poly(I:C) treatment. Bars represent the mean ± SEM. Significance was calculated using two-way analysis of variance. n.s. denotes not significant (6–8 mice/group). (B) Percentages of CMPs, GMPs, and MEPs in BM, DP, and PB of poly(I:C)-treated mice and non-treated mice at 24 h after acute poly(I:C) treatment. Bars represent the mean ± SEM. Significance was calculated using two-way analysis of variance. ∗ denotes P < 0.05, ∗∗ denotes P < 0.01 and n.s. denotes not significant (6–12 mice/group). (C) Percentage of immune cells in BM, DP, and PB of poly(I:C)-treated mice and non-treated mice at 24 h after chronic poly(I:C) treatment. Bars represent the mean ± SEM. Significance was calculated using two-way analysis of variance. ∗∗ denotes P < 0.01, and n.s. denotes not significant (6 mice/group, n = 12). (D) Percentage of immune cells in BM, DP, and PB of poly(I:C)-treated mice and non-treated mice at 24 h after acute poly(I:C) treatment. Bars represent the mean ± SEM. Significance was calculated using two-way analysis of variance. ∗ denotes P < 0.05, ∗∗ denotes P < 0.01, and n.s. denotes not significant (6 mice/group, n = 12).