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. 2022 Feb 14;21:51. doi: 10.1186/s12943-022-01521-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Downregulation of METTL14 was associated with poor prognosis in GC. A TCGA assessment of the expression levels of METTL14 in paired and unpaired GC tissues. B, C Western blot analysis of the protein expression of METTL14 in 10 paired GC tissue samples. D IHC analysis of the protein expression of METTL14 in 90 paired GC tissue samples. E Tissue microarray as well as TCGA and GSE22377 cohorts analysis of the association of METTL14 with the prognosis of GC patients