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. 2022 Feb 14;21:51. doi: 10.1186/s12943-022-01521-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Knockdown of METTL14 promoted proliferation and invasion of GC. A RT-qPCR and Western blot analysis of the mRNA and protein expression levels of METTL14 in GSE1 and GC cell lines. B RT-qPCR and Western blot analysis of the transfection efficiency of si-METTL14 in MGC-803 and AGS cells or METTL14 in SGC-7901 cells. C MTT analysis of the effects of METTL14 knockdown or overexpression on the GC cell viability. D Colony formation analysis of the effects of METTL14 knockdown or overexpression on the colony formation ability. E Transwell analysis of the effects of METTL14 knockdown or overexpression on the GC cell invasion. Data are the means ± SEM of three experiments. *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001.