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. 2022 Feb 15;21:15330338221077803. doi: 10.1177/15330338221077803

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© The Author(s) 2022

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — SLC16A1-AS1 acts as a sponge of miR-143-3p. (A, B) The localization of SLC16A1-AS1 was examined in 786-O and Caki-1 cells. (C) The putative binding site of miR-143-3p on SLC16A1-AS1. (D) The expression level of miR-143-3p in RCC cells was determined by qRT–PCR. (E) MS2-based RIP assay in RCC cells. (F) The expression level of miR-143-3p in RCC cells after transfection with si-SLC16A1-AS1. *p<.05, **p<.01 compared with the NC group.

Abbreviations: miR-143-3p, microRNA-143-3p; RCC, renal cell carcinoma; qRT–PCR, quantitative reverse transcription–polymerase chain reaction; RIP, RNA-binding protein immunoprecipitation; NC, negative control.