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. 2022 Feb 7;9:781327. doi: 10.3389/fcell.2021.781327

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Copyright © 2022 Zhao, Mao, Zhu, Zou, Peng, Yang, Li, Li, Ge and Cui.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Current known candidate genes of ASD involved in GABAergic synaptic transmission and related mechanisms. Current candidate genes induce dysfunction of GABAergic transmission by affecting transcription of GABA-A subunits, presynaptic GABA release, formation of GABAergic synapse, and synaptic structure–mediated transmission. GABA-related candidate genes and regulatory role are represented by red letters. Pharmacological modulators of GABA-R reported to have alleviation efficacy are represented by blue letters.