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. 2022 Feb 21;53:13. doi: 10.1186/s13567-022-01030-3

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Expression of cell cycle promoter CDK1. CDK4 gene expression in fetal tissues including the heart (HRT), kidney (KID), spleen (SPLN), liver (LVR), lung (LNG), thymus (THY), and loin muscle (MUS) derived from fetuses collected from PRRSV-2 challenged dams at 21 days post-inoculation and classified based on viral load in serum and thymus as uninfected (UNIF, n = 10), high viral load viable (HV-VIA, n = 10) or high viral load meconium stained (HV-MEC, n = 10), or from control fetuses (CON n = 10) collected from gestation day matched non-inoculated control gilts. Fold changes were calculated within tissue relative to the average of the CON group with different superscripts denoting statistical differences (P < 0.05) within a given tissue.