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. 2022 Jan 21;50(3):1335–1350. doi: 10.1093/nar/gkac024

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© The Author(s) 2022. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 8. — Alzheimer's disease GWAS variants analysis. (A) Variant-gene network of the two variants rs7207400 and rs199499. (B) Effect of the variant rs7207400 on the transcript level of LRRC37A2 gene under the additive model. (C) Effect of the variant rs199499 on the transcript level of LRRC37A2 gene under the additive model. (D) Combined effect of the two variants rs7207400 (first pair of nucleotides in the label) and rs199499 (second pair) on LRRC37A2. (E) Variant-gene network of the community formed by variants rs7225151, rs113260531 and rs80257887. (F) Effect of the variant rs7225151on the transcript level of HIST2H2AC gene under the recessive model. (G) Effect of the variant rs80257887 on the transcript level of HIST2H2AC gene under the dominant model. (H) Combined effect of the two variants rs7225151(first pair of nucleotides in the label) and rs80257887 (second pair) on HIST2H2AC.