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. 2022 Feb 11;25(4):123. doi: 10.3892/mmr.2022.12639

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Copyright: © Liang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Knockdown of DDX11-AS1 enhances the sensitivity of drug-resistant breast cancer cells to PTX. Reverse transcription-quantitative PCR assay detected the expression levels of DDX11-AS1 in (A) MCF-7/PTX, (B) MDA-MB-231/PTX, (C) MCF-7 and (D) MDA-MB-231 cells after transfection. CCK-8 assay detected (E) cell viability and (F) cell inhibition rate in the MCF-7/PTX cell line. CCK-8 assay detected (G) cell viability and (H) cell inhibition rate in the MDA-MB-231/PTX cell line. ***P<0.001 vs. shRNA-NC. CCK-8, Cell Counting Kit-8; DDX11-AS1, DDX11 antisense 1; lncRNA, long non-coding RNA; NC, negative control; PTX, paclitaxel; shRNA, short hairpin RNA.