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. 2022 Feb 11;25(4):123. doi: 10.3892/mmr.2022.12639

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Copyright: © Liang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 8. — Knockdown of DDX11-AS1 reduces the sensitivity of drug-resistant breast cancer cells to PTX by targeting the upregulation of miR-497. RT-qPCR assay detected the expression of miR-497 in (A) MCF-7/PTX and (B) MDA-MB-231/PTX cells after transfection. ***P<0.001 vs. Inhibitor-NC. CCK-8 assay detected (C) cell viability and (D) cell inhibition rate in the MCF-7/PTX cell line. CCK-8 assay detected (E) cell viability and (F) cell inhibition rate in the MDA-MB-231/PTX cell line. ***P<0.001 vs. Control; ^##P<0.01 vs. shRNA-DDX11-AS1 + inhibitor-NC. DDX11-AS1, DDX11 antisense 1; miR-497, microRNA-497; NC, negative control; PTX, paclitaxel; shRNA, short hairpin RNA.