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. 2022 Feb 23;11:7. doi: 10.1186/s40164-022-00260-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — The effects of HOXA13 on angiogenesis, migration, and invasion in GC cells. a–d Angiogenesis ability of HOXA13 overexpression or knockdown stable cell lines was detected by tube formation assay and could be partly rescued by transfection with siFN1 or FN1 overexpression plasmid. Original magnification, ×200. e–h Migration and invasion ability of HOXA13 overexpression or knockdown stable cell lines were tested by Transwell assay and could be partly rescued by transfection with siFN1 or FN1 overexpression plasmid. Original magnification, ×200. i The protein expression of EMT-related markers was detected by Western blotting (**p < 0.01, ***p < 0.001)