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. 2022 Feb 9;9:778489. doi: 10.3389/fmed.2022.778489

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Copyright © 2022 Maccio, Zinkernagel, Schuepbach, Probst-Mueller, Frontzek, Brugger, Hofmaenner, Moch and Varga.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Representative histopathological findings in autopsies from the second wave, illustrating examples of coinfections in the lung of the patients with coronavirus disease 2019 (COVID-19). (A) Lung tissue with aspergillosis (arrow) and surrounding acute inflammation (H&E, magnification 10 x). (B) Details of image A showing the typical hyphae of Aspergillus spp. (PAS, magnification 30 x). (C) Acute (bacterial) bronchopneumonia showing granulocytic exudate in the alveolar space and destruction of alveolar septa (H&E, magnification 6 x); the inset illustrates granulocytic inflammation with destruction of a septum (magnification 50 x). (D) Herpes simplex pneumonia exhibiting typical herpes-associated nuclear changes (molding, multinucleation, margination of chromatin, see the arrow) (H&E, magnification 25 x). (E) Immunohistochemistry for Herpes simplex virus demonstrates a granular cytoplasmic and nuclear positivity along with the typical nuclear changes (magnification 25 x).