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. 2022 Feb 16;2022:8603427. doi: 10.1155/2022/8603427

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Copyright © 2022 Zhang Yang et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

PMC Copyright notice

CPE-derived EVs deliver miR-155-5p to aggravate brain injury in mice. (a) miR-155-5p expressions in mouse brain tissues were estimated using RT-qPCR. (b) The mass ratio of the ipsilateral (right) hemisphere to the contralateral (left) hemisphere of the brain in tMCAO model mice was calculated. (c) The neurological function of mice was determined by mNSS. (d) Cerebral infarctions in tMCAO model mice were detected by TTC. (e) Immunoblotting for autophagy-related protein levels in mouse brain tissues. ^∗p < 0.05 vs. sham-operated mice. ^#p < 0.05 vs. EV- and agomir-NC-cotreated mice. ^&p < 0.05 vs. mice cotreated with EVs and antagomir-NC (n = 3).