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. 2022 Jan 11;39(1):239–250. doi: 10.1007/s10815-022-02395-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — The expression levels of FPR2 were significantly increased in the placenta tissues of patients with PE. (A) The expression levels of FPR2 in placenta tissues from CT (n = 10) and patients with PE (n = 10) were detected using immunohistochemistry analysis. Scale bar, 50 μm. (B) The expression levels of FPR2 were determined using western blotting analysis (***P < 0.001: PE vs CON group). (C) The expression levels of FPR2 in response to treatment of HTR8/SVneo cells with different concentrations of LPS (0, 10, 50 and 100 ng/mL, 1 and 5 μg/mL) (***P < 0.001: LPS vs CON group). (D) The expression levels of FPR2 following different durations of treatment (0, 3, 6, 12 and 24 h) in the presence of 100 ng/ml LPS (**P < 0.01, ***P < 0.001: LPS vs CON group)