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A hypothetical model for how IAV utilizes TRIM34 to promote the NLRP3 inflammasome, NF-κB, and programmed cell death pathways by ubiquitination of ZBP1. IAV, influenza A virus; IL, interleukin; NF-κB, nuclear factor κB; TRIM, tripartite motif; ZBP1, Z-DNA-binding protein 1.
