Skip to main content
. 2022 Feb 23;18:17. doi: 10.1186/s13223-022-00658-2

Table 2.

Clinical laboratory results in twin females with PD

Test Patient 1 Patient 2 Normal range
Blood cell counts
 White blood cells 7.26 8.13 4.19–9.43 10e9/L
 Neutrophils 2.2 4.22 1.82–7.47 10e9/L
 Lymphocytes 4.09 (H) 2.9 1.16–3.33 10e9/L
 Monocytes 0.73 (H) 0.76 (H) 0.19–0.72 10e9/L
 Plateletsa 118 (L) 154 130–400 10e9/L
 Mean platelet volume 9.8 10.3 8–12 fL
 Hemoglobin 130 149 105–150 g/L
Immunoglobulins
 IgA 0.81 1.35 0.52–1.92 g/L
 IgM 0.63 1.07 0.47–3.11 g/L
 IgEb 423 568 <629 kU/L
 IgGc 8.92 9.56 7–15.9 g/L
 IgG1 5.77 7.54 3.15–8.55 g/L
 IgG2 0.30 (L) 0.65 0.64–4.95 g/L
 IgG3 0.3 0.29 0.23–1.96 g/L
 IgG4 0.158 0.384 0.11–1.57 g/L
Specific antibody titers to diphtheria, tetanus, pneumococcal, rubella, varicella, and measles vaccine antigens Good, sustained, protective antibody titers to vaccines except borderline measles IgG response Good, sustained, protective antibody titers to all vaccines
B cell proliferation (% of CpG-stimulated cells divided) 68.1% 65.0% 63.2–100%d
T cell proliferation: mitogen and antigen stimulation of PBMCs by PHA, ConA, PWM, anti-CD3, anti-CD3 + IL-2, IL-2, tetanus toxoid, diphtheria toxoid, and Candida albicans antigens Normal lymphocyte proliferation to all mitogens and antigens tested Normal lymphocyte proliferation to all mitogens and antigens tested
NK cell function
 NK cell cytotoxicity (NK cell killing activity) Normal Normal
 Degranulation (CD107a+) 27% 22% 11–35%
 Neutrophils NBT reduction Normal oxidative burst of 99% Normal oxidative burst of 100%
Complement
 CH50 classical 93 70 42–96 U/mL
 C3e 1.54 1.63 1.1–1.8 g/L
 C4e 0.26 0.24 0.17–0.39 g/L
 MBL 0 (L) 0 (L) 30–200%
 Alternate complement 119 107 >40%
Inflammatory markers
 ESR 18 (H) 2 0–9 mm/h
 CRP 24.6 (H) 6 (H) 0–5.0 mg/L
 Ferritin 400 (H) 2006 (H) 5.5–67 mcg/L
 IL-18  > 36,600 (H) 28,803 (H) <266 pg/mL
 SAA 9837 8511 ng/mL, within 30–70% of normal
Auto-antibodiesf
 ANA ANA IIF + (1:320, speckled pattern) All negative
ENA, anti-TTG-IgA, anti-cardiolipin, anti-B2GP1, LA, ASMA, APCP, anti-LKM, anti-PR3, anti-MPO, anti-CCP, RF, anti-TPO anti-TPO + 
Lipid profile
 LDL NDg 3.08

Acceptable<2.85

H>3.36 mmol/L

 HDL 0.79 (L) 1.2 Acceptable>1.17 mmol/L
 TGA 6.1 (H) 2.21 (H)

Acceptable<1.02

H>1.46 mmol/L

 Cholesterol 6.86 (H) 5.28 (H)

Acceptable<4.40

H>5 mmol/L

L, low; H, high; PHA, phytohemagglutinin; ConA, concanavalin A; PWM, pokeweed mitogen; NK, natural killer cells; MBL, mannose-binding lectin; ESR, erythrocyte sedimentation rate; CRP, C-reactive protein; SAA, serum amyloid A; pc, percentile; ANA, antinuclear antibodies; ENA, extractable nuclear antigen antibodies; SS-A/B, Sjögren's-syndrome-related antigen A/B autoantibodies; anti-TTG-IgA, anti-tissue transglutaminase IgA antibodies; anti-B2GP1, anti-beta-2-glycoproteins antibodies; ASMA, anti-smooth muscle antibodies; ACPA, anti-citrullinated protein antibodies; anti-LKM, anti-liver-kidney-muscle antibodies; anti-MPO, anti-myeloperoxidase antibodies; anti-CCP, anti-cyclic citrullinated peptide antibodies; RF, rheumatoid factor; anti-TPO, anti-thyroid peroxidase antibodies; LDL, low-density lipoprotein; HDL, high-density lipoprotein; TGA, triglycerides

aThrombocytopenia reported in literature [1]

bIncreased serum IgE levels reported in literature [3]

cHypergammaglobulinemia reported in literature [2]

dMean ± 2SD, n = 6 healthy donors

eHypocomplementemia (C3 and C4) reported in literature [3]

fPositive ANA, anti-dsDNA, anti-ENA (anti-Ro), anti-Sm, and anti-chromatin have been found in individuals with prolidase deficiency even in the absence of clinical findings of SLE [2]

gNot determined due to high TGA