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. 2021 Sep 17;26(12):7346–7354. doi: 10.1038/s41380-021-01272-1

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A Top row, brain maps of t-statistics representing strength of association between CRP and functional connectivity of the hippocampus (with each of the other brain regions). Bottom row, cortical maps highlighting regions where CRP was significantly, positively correlated with functional connectivity with the hippocampus (P_FDR < 0.05). B Scatterplots illustrating the relationship between CRP and hippocampus connectivity for two cortical regions: POS1 and v23ab. Bottom, scatterplots of CRP versus PD for the same cortical regions; * denotes P < 0.05. C Voxel based analysis of functional connectivity dependence on CRP for a seed in the PCC (top row) and for a seed in the right Hippocampus (bottom row). Functional connectivity between hippocampus, PCC, and mPFC was positively correlated with CRP (only significant clusters with α < 0.05 are shown; global significance threshold P < 0.05).