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. 2022 Feb 24;13(2):179. doi: 10.1038/s41419-022-04630-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A, B SF3B4 knockdown and overexpression efficiency was confirmed by qRT-PCR (n = 3 biologically independent samples) and western blotting. C Growth curve showed the growth of ovarian cancer cells upon SF3B4 knockdown and overexpression (n = 3 biologically independent samples). D EdU assay showed the proliferation ability of ovarian cancer cells after SF3B4 knockdown and overexpression (n = 3 biologically independent samples). E The effect of SF3B4 on colony formation in ovarian cancer cells (n = 3 biologically independent samples). F, G In HEY and SKOV3 cells, up and downregulation of SF3B4 affected migration and invasion capacity (n = 3 biologically independent samples). P-value was obtained by Unpaired t-test. Results represent the mean ± SD of three independent experiments. *P < 0.05, **P < 0.01.