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. 2022 Feb 24;13(2):179. doi: 10.1038/s41419-022-04630-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — A, B RAD52 knockdown efficiency was confirmed by qRT-PCR (n = 3 biologically independent samples) and western blotting. C Growth curve and (D) EdU assay and (E) clonogenic assay were used to evaluate the effect of SF3B4 on the proliferation of HEY, A2780, and SKOV3 cells (n = 3 biologically independent samples). F Transwell assay showed the invasion capacity of ovarian cancer cells upon RAD52 knockdown (n = 3 biologically independent samples). G, H Different growth and invasion abilities associated with different expression levels of SF3B4 and RAD52 in SKOV3 cells (n = 3 biologically independent samples). I Growth curve assay was used to investigate the potential of RAD52 to rescue the loss of SF3B4 in ovarian cancer cells (n = 3 biologically independent samples). P-value was obtained by Unpaired t-test. *P < 0.05, **P < 0.01.